Chondroitin sulfate reliable treatment option for diabetic osteoporosis patients
A new study by Shan Shan Qi and the team revealed chondroitin sulfate could prevent streptozotocin (STZ)-induced diabetic osteoporosis (DOP) and repair bone microstructure. The main mechanism is through regulating bone metabolism, anti-oxidation, and anti-inflammatory. The findings of the study, published in the Frontiers in Endocrinology, indicate that chondroitin sulfate could be used for the development of anti-DOP functional foods and diet interventions for diabetes.
The results showed that chondroitin sulfate can reduce blood sugar and relieve symptoms in diabetic rats. In addition, it can significantly increase bone mineral density, reduce bone marrow adipocyte number in diabetic rats, and improve bone microstructure. After 10 weeks of chondroitin sulfate administration, the SOD activity level was upregulated, as well as CAT levels, indicating that chondroitin sulfate can alleviate oxidative stress in diabetic rats.
For more details check out the full story in link below:
Chondroitin Sulfate Reliable Treatment For Diabetic Osteoporosis: Study
Disclaimer: This website is primarily for healthcare professionals. The content here does not replace medical advice and should not be used as medical, diagnostic, endorsement, treatment, or prescription advice. Medical science evolves rapidly, and we strive to keep our information current. If you find any discrepancies, please contact us at corrections@medicaldialogues.in. Read our Correction Policy here. Nothing here should be used as a substitute for medical advice, diagnosis, or treatment. We do not endorse any healthcare advice that contradicts a physician's guidance. Use of this site is subject to our Terms of Use, Privacy Policy, and Advertisement Policy. For more details, read our Full Disclaimer here.
NOTE: Join us in combating medical misinformation. If you encounter a questionable health, medical, or medical education claim, email us at factcheck@medicaldialogues.in for evaluation.