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Visual Display Terminal may induce Dry Eye Disease through both direct and indirect routes

MD BureauWritten by MD Bureau Published On 2022-04-26T09:00:21+05:30  |  Updated On 2022-04-26T09:00:25+05:30
Visual Display Terminal may induce Dry Eye Disease through both direct and indirect routes

Dry eye disease (DED) is a highly prevalent condition affecting several hundred million people worldwide. Visual display terminal (VDT) use is an important risk factor for DED. A recent study suggests that decreased blink rates and increased incomplete blinks contribute to visual display terminal associated dry eye disease. The study findings were published in the Acta Ophthalmologica on...

Dry eye disease (DED) is a highly prevalent condition affecting several hundred million people worldwide. Visual display terminal (VDT) use is an important risk factor for DED.

A recent study suggests that decreased blink rates and increased incomplete blinks contribute to visual display terminal associated dry eye disease. The study findings were published in the Acta Ophthalmologica on April 19, 2022.

VDT use has been shown to decrease TBUT and increase interblink interval. However, much is still unknown regarding the adverse effects of VDT use on the ocular surface. Therefore, Dr Ketil Fjærvoll and his team conducted a review on pathophysiological mechanisms promoting VDT-associated DED.

In this review, the researchers searched PubMed for literature investigating the relationship between dry eye and VDT. They evaluated the relevance to the pathophysiology of DED. They included a total of fifty-five articles and examined the pathophysiological mechanisms and multiple hypotheses.

Key findings of the study:

  • The researchers noted that visual display terminal (VDT) use causes DED mainly through impaired blinking patterns.
  • Although some studies reported changes in parasympathetic signalling and increased exposure to blue light, which could disrupt ocular homeostasis, they noted that the studies lack sufficient scientific support.
  • They highlighted that these changes may lead to a reduced function of the tear film, lacrimal gland, goblet cells and meibomian glands, all contributing to DED development.

The authors concluded, "Visual display terminal (VDT) use appears to induce DED through both direct and indirect routes. Decreased blink rates and increased incomplete blinks increase the exposed ocular evaporative area and inhibit lipid distribution from meibomian glands."

They further added, "Although not adequately investigated, changes in parasympathetic signalling may impair lacrimal gland and goblet cell function, promoting tear film instability. More studies are needed to better target and improve the treatment and prevention of VDT-associated DED".

For further information:

DOI: https://doi.org/10.1111/aos.15150


DED dry eye disease DED pathophysiology tear film VDT VDT-associated dry eye lacrimal gland goblet cells meibomian glands Acta Ophthalmologica 
Article Source :  Acta Ophthalmologica
MD Bureau
MD Bureau

    Medical Dialogues Bureau consists of a team of passionate medical/scientific writers, led by doctors and healthcare researchers.  Our team efforts to bring you updated and timely news about the important happenings of the medical and healthcare sector. Our editorial team can be reached at editorial@medicaldialogues.in.

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